Items where authors include "Brown, C.J."

Export as [feed] Atom [feed] RSS
Jump to: Article
Number of items: 8.

Article

Brown, C.J., McGinty, J., Islam, M.T. et al. (19 more authors) (2024) Integrated continuous process design for crystallisation, spherical agglomeration, and filtration of lovastatin. Journal of Pharmaceutical Innovation, 19. 9. ISSN 1872-5120

Hany, U. orcid.org/0000-0002-4486-1625, Watson, C.M. orcid.org/0000-0003-2371-1844, Liu, L. et al. (11 more authors) (2024) Novel Ameloblastin Variants, Contrasting Amelogenesis Imperfecta Phenotypes. Journal of Dental Research, 103 (1). pp. 22-30. ISSN 0022-0345

Hany, U. orcid.org/0000-0002-4486-1625, Watson, C.M. orcid.org/0000-0003-2371-1844, Liu, L. et al. (11 more authors) (2024) Novel ameloblastin variants, contrasting amelogenesis imperfecta phenotypes. Journal of Dental Research, 103 (1). pp. 22-30. ISSN 0022-0345

Hany, U. orcid.org/0000-0002-4486-1625, Watson, C.M. orcid.org/0000-0003-2371-1844, Liu, L. et al. (17 more authors) (2023) Heterozygous COL17A1 variants are a frequent cause of amelogenesis imperfecta. Journal of Medical Genetics. ISSN 0022-2593

Hany, U. orcid.org/0000-0002-4486-1625, Watson, C.M. orcid.org/0000-0003-2371-1844, Liu, L. et al. (17 more authors) (2023) Heterozygous COL17A1 variants are a frequent cause of amelogenesis imperfecta. Journal of Medical Genetics. ISSN 0022-2593

Brown, C.J., Webb, T.L. orcid.org/0000-0001-9320-0068, Robinson, M.A. et al. (1 more author) (2019) Athletes’ retirement from elite sport: A qualitative study of parents and partners’ experiences. Psychology of Sport and Exercise, 40. pp. 51-60. ISSN 1469-0292

Brown, C.J., Webb, T.L. orcid.org/0000-0001-9320-0068, Robinson, M.A. et al. (1 more author) (2018) Athletes' experiences of social support during their transition out of elite sport: An interpretive phenomenological analysis. Psychology of Sport and Exercise, 36. pp. 71-80. ISSN 1469-0292

Brookes, S.J., Barron, M.J., Smith, C.E.L. et al. (7 more authors) (2017) Amelogenesis imperfecta caused by N-terminal enamelin point mutations in mice and men is driven by endoplasmic reticulum stress. Human Molecular Genetics, 26 (10). pp. 1863-1876. ISSN 0964-6906

This list was generated on Sat May 4 08:58:44 2024 BST.