Caveolin1 interacts with the glucocorticoid receptor in the lung but is dispensable for its anti-inflammatory actions in lung inflammation and Trichuris Muris infection

Caratti, G, Poolman, T, Hurst, RJ et al. (8 more authors) (2019) Caveolin1 interacts with the glucocorticoid receptor in the lung but is dispensable for its anti-inflammatory actions in lung inflammation and Trichuris Muris infection. Scientific Reports, 9. 8581. ISSN 2045-2322

Abstract

Metadata

Authors/Creators:
  • Caratti, G
  • Poolman, T
  • Hurst, RJ
  • Ince, L
  • Knight, A
  • Krakowiak, K
  • Durrington, HJ
  • Gibbs, J
  • Else, KJ
  • Matthews, LC
  • Ray, DW
Copyright, Publisher and Additional Information: © The Author(s) 2019. This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.
Keywords: Glucocorticoid; Caveolin; Inflammation; Asthma
Dates:
  • Published: 12 June 2019
  • Accepted: 24 May 2019
  • Published (online): 12 June 2019
Institution: The University of Leeds
Academic Units: The University of Leeds > Faculty of Medicine and Health (Leeds) > School of Medicine (Leeds) > Leeds Institute of Cancer and Pathology (LICAP) > Infection and Immunity (Leeds)
Depositing User: Symplectic Publications
Date Deposited: 24 Jun 2019 11:17
Last Modified: 24 Jun 2019 11:17
Status: Published
Publisher: Nature Publishing Group
Identification Number: https://doi.org/10.1038/s41598-019-44963-0
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