Overexpression of Desmoglein 2 in a mouse model of Gorlin syndrome enhances spontaneous basal cell carcinoma formation through STAT3-mediated Gli1 expression

Brennan-Crispi, DM, Overmiller, AM, Tamayo-Orrego, L et al. (11 more authors) (2019) Overexpression of Desmoglein 2 in a mouse model of Gorlin syndrome enhances spontaneous basal cell carcinoma formation through STAT3-mediated Gli1 expression. Journal of Investigative Dermatology, 139 (2). pp. 300-307. ISSN 0022-202X

Abstract

Metadata

Authors/Creators:
  • Brennan-Crispi, DM
  • Overmiller, AM
  • Tamayo-Orrego, L
  • Marous, MR
  • Sahu, J
  • McGuinn, KP
  • Cooper, F
  • Georgiou, IC
  • Frankfurter, M
  • Salas-Alanis, JC
  • Charron, F
  • Millar, SE
  • Mahoney, MG
  • Riobo-Del Galdo, NA https://orcid.org/0000-0002-8942-7873
Copyright, Publisher and Additional Information: © 2018 The Authors. Published by Elsevier, Inc. on behalf of the Society for Investigative Dermatology. This is an author produced version of a paper published in Journal of Investigative Dermatology. Uploaded in accordance with the publisher's self-archiving policy.
Keywords: basal cell carcinoma; hedgehog; stat3; desmoglein; signalling
Dates:
  • Published: February 2019
  • Accepted: 4 September 2018
  • Published (online): 3 October 2018
Institution: The University of Leeds
Academic Units: The University of Leeds > Faculty of Medicine and Health (Leeds) > Institute of Molecular Medicine (LIMM) (Leeds) > Section of Experimental Oncology (Leeds)
The University of Leeds > Faculty of Medicine and Health (Leeds) > School of Medicine (Leeds) > Leeds Institute of Cancer and Pathology (LICAP) > Section of Experimental Oncology (Leeds)
Depositing User: Symplectic Publications
Date Deposited: 19 Sep 2018 10:43
Last Modified: 03 Oct 2019 00:38
Status: Published
Publisher: Elsevier
Identification Number: https://doi.org/10.1016/j.jid.2018.09.009

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