Loss of the homeostatic protein BPIFA1, leads to exacerbation of otitis media severity in the Junbo mouse model

Mulay, A., Hood, D.W., Williams, D. et al. (5 more authors) (2018) Loss of the homeostatic protein BPIFA1, leads to exacerbation of otitis media severity in the Junbo mouse model. Scientific Reports, 8. 3128. ISSN 2045-2322

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Copyright, Publisher and Additional Information: © The Author(s) 2018. This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. Te images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.
Dates:
  • Published: 15 February 2018
  • Accepted: 30 January 2018
Institution: The University of Sheffield
Academic Units: The University of Sheffield > Faculty of Medicine, Dentistry and Health (Sheffield) > School of Dentistry (Sheffield)
The University of Sheffield > Faculty of Medicine, Dentistry and Health (Sheffield) > Department of Cardiovascular Science (Sheffield)
The University of Sheffield > Sheffield Teaching Hospitals
Funding Information:
FunderGrant number
BIOTECHNOLOGY AND BIOLOGICAL SCIENCES RESEARCH COUNCIL (BBSRC)BB/K009737/1
Depositing User: Symplectic Sheffield
Date Deposited: 27 Feb 2018 11:59
Last Modified: 01 Nov 2018 10:24
Published Version: https://doi.org/10.1038/s41598-018-21166-7
Status: Published online
Publisher: Nature Publishing Group
Refereed: Yes
Identification Number: https://doi.org/10.1038/s41598-018-21166-7
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