Post translational changes to α-synuclein control iron and dopamine trafficking; a concept for neuron vulnerability in Parkinson’s disease

Duce, JA orcid.org/0000-0002-8762-2245, Wong, BX, Durham, H et al. (3 more authors) (2017) Post translational changes to α-synuclein control iron and dopamine trafficking; a concept for neuron vulnerability in Parkinson’s disease. Molecular Neurodegeneration, 12. 45. ISSN 1750-1326

Abstract

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Authors/Creators:
Copyright, Publisher and Additional Information: © The Author(s) 2017. Open Access. This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
Keywords: α-synuclein; Iron; Dopamine; Endosomal trafficking; Oxidative stress; Post translational modification; N-terminal acetylation; Phosphorylation; Oxidation
Dates:
  • Published: December 2017
  • Accepted: 2 June 2017
  • Published (online): 7 June 2017
Institution: The University of Leeds
Academic Units: The University of Leeds > Faculty of Biological Sciences (Leeds) > School of Biomedical Sciences (Leeds)
Depositing User: Symplectic Publications
Date Deposited: 09 Jun 2017 14:48
Last Modified: 09 Jun 2017 14:48
Status: Published
Publisher: BioMed Central
Identification Number: https://doi.org/10.1186/s13024-017-0186-8

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