Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.

Nassar, M.A., Levato, A., Stirling, L.C. et al. (1 more author) (2005) Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8. Molecular Pain, 1. 24. ISSN 1744-8069

Abstract

Metadata

Authors/Creators:
  • Nassar, M.A.
  • Levato, A.
  • Stirling, L.C.
  • Wood, J.N.
Copyright, Publisher and Additional Information: © 2005 Nassar et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Keywords: Animals; Behavior, Animal; Female; Gene Expression Regulation, Developmental; Inflammation; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nerve Tissue Proteins; Nociceptors; Pain; Pain Threshold; Potassium Channels, Voltage-Gated; Sex Factors; Shaker Superfamily of Potassium Channels
Dates:
  • Published: 22 August 2005
  • Accepted: 22 August 2005
Institution: The University of Sheffield
Academic Units: The University of Sheffield > Faculty of Science (Sheffield) > School of Biological Sciences (Sheffield) > Department of Biomedical Science (Sheffield)
Depositing User: Symplectic Sheffield
Date Deposited: 13 Jan 2017 13:02
Last Modified: 23 Jun 2017 15:14
Published Version: https://doi.org/10.1186/1744-8069-1-24
Status: Published
Publisher: SAGE Publications
Refereed: Yes
Identification Number: https://doi.org/10.1186/1744-8069-1-24
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